Geert Vanden Bossche's latest report; note he says what I have been saying about damage to INNATE immune system by COVID vax; I confess, I learn lots from him so must give him credit for my learning

by Paul Alexander

He is incredibly bright, tops...him and Bridle. I share the full report link but pull out this sage piece below & the bolded words as to my repeated sharing but as said above, I stand on Geert's depth

Geert continues to warn us and we must take heed and note, for his discussion and insight is optimal and there is sage in his words; we are indeed playing with fire if we do not stop the vaccine program.

Source: Geert

‘What does the official ‘expert’ narrative tell us? Despite all biological evidence, including Darwinian theory, global and public health officials and advising experts have continued to dismiss serious warnings of myself and others that the evolutionary dynamics of this pandemic may largely be shaped by the mass vaccination program they have been advocating for all along. But now that the virus (i.e., Omicron) has become largely resistant to the vaccine-induced neutralizing Abs, even some independent scientists tend to believe that C-19 vaccinations have contributed to reducing the severity of this pandemic and that we’re now shifting to a ‘natural’ mass immunization mode that is even better because it uses a highly infectious but overwhelmingly mild virus (i.e., Omicron). Omicron would, therefore, serve as an almost perfect ‘live attenuated vaccine’! Stakeholders of this thoughtless vaccination program are now claiming that we owe this ‘blessing’ to the success of this very program. Anyway, many scientists now seem to believe that - thanks to Omicron - the population managed to open a window of opportunity for establishing herd immunity and, therefore, to end the pandemic!

What does the epidemiology of the current pandemic teach us? There are currently several observations in highly vaccinated populations that are suspicious of suppression of the innate immune system in most of the population (i.e., vaccinees) in that they are pointing to enhanced susceptibility of the vaccinees to infection instead of diminished susceptibility, which would be expected if herd immunity were growing. Extension of the mass vaccination program to very young and poorly antigen-experienced children will only enlarge the reservoir of vulnerable individuals and cause additional vaccine breakthrough surges in this age group. Because of the high level of Omicron’s infectiousness in highly vaccinated populations, the frequency of infections occurring at any given time has dramatically increased. It is important, though, to note that the number of ‘cases’ is no longer a reliable metric for the infection rate in the population as cases now almost exclusively relate to cases of C-19 disease.

As the decline in hospitalization and death rates have now led governments and public health authorities to largely lift the public health measures and as many ‘cases’ are now going unreported because infections are mostly characterized by relatively mild symptoms (both in the unvaccinated and vaccinated part of the population), the overall number of cases - as reported by the official national and international databases - is certainly largely underestimated4 . Nevertheless, many countries are still reporting overall infection rates that are relatively high.

This observation together with the moderate decline that follows a peak of cases and/or the rapid succession of individual waves that are only separated by short intervals and/or failure of the case rate curves to rejoin the baseline in-between two waves, are all signs of the population’s enhanced susceptibility to infection (see fig. 1). So, if the vaccines would perform as well as our natural immune defense during a natural pandemic, we would expect most people to develop asymptomatic infection (instead of mild or moderate disease) and that after each of a limited number of waves, the infection rate sharply declines to rejoin the baseline (instead of leveling off prematurely before temporarily transitioning into a kind of plateau or irregular pattern of smaller waves). However, high waves of infectiousness are no longer a guarantee of a spectacular subsequent decline of cases. It seems, therefore, that the fluctuation in the case rates is merely reflecting changes in the susceptibility to infection in different parts of the population with no evidence of capacity being built to provide sterilizing immunity.

This is what is now causing ‘herd susceptibility’ instead of ‘herd immunity’. The more the mass vaccination campaigns are now involving younger and younger age groups, the more the reservoir of susceptible people is growing and the more surges will likely be grafted on the new baseline, the level of which has already been raised following waves of more infectious variants.

It has been reported that vaccinees are, indeed, more susceptible to infection but that this enhanced susceptibility does not translate in more (cases of) severe disease (https://www.medrxiv.org/content/10.1101/2022.01.28.22270044v1). It seems, therefore, as if the vaccine is responsible for promoting the vaccinee’s susceptibility to infection while hampering progression of infection to severe disease. This is in sharp contrast to the course of a natural pandemic in an unvaccinated population, in which waves of infection are associated with a substantial surge in morbidity and mortality, typically in the most vulnerable part of the population.

These surges are typically followed by a dramatic reduction of the infection rate and it typically only takes a few waves for a natural pandemic to transition into endemicity as this is what it takes to protect the remaining vulnerable part of the population by herd immunity. Based on the evolutionary dynamics of the current pandemic and the fact that the above-described trends are most obvious in highly vaccinated populations, it is tempting to postulate that the overall population (the majority of which consists of vaccinees) exerts immune pressure on the virus such as to reduce the severity of C-19 disease while enabling a high level of susceptibility to infection. It is highly unlikely that the combination of these 2 characteristics would be pure coincidence. It is more likely that they have a common denominator which is to be sought in the large-scale immune intervention on the population.’…