Immunity against Omicron from breakthrough infection could be a matter of timing, NATURE; if you go back to prior posts you would see I/we (Geert) said this, we were describing the data

by Paul Alexander

This NATURE piece mirrors what we have been saying, that aggressive rapid vaccination, with no space between vaccines, causes massive infections; antibodies not mature, developed yet

We mean those nations who have had high levels of vaccination aggressively, while the virus is circulating, that ‘infectious pressure’ we keep referring to, then the immune system, the mounting vaccinal antibodies have not had time to mature and develop, and this is where the breakthrough happens. Infectious pressure against mounting sub-optimal immune pressure…I have been writing this repeatedly for a reason and now se this publication…I learn from GVB, the master. Also, it is the aggressive repeated shots and boosters that is the problem…

We also theorize that the innate antibodies in the unvaccinated are getting ‘trained’ and ‘learning’ due to the ongoing infectious pressure and are able to withstand the infectious pressure…what does this mean? leave them alone…again, it is the vaccination using a non-sterilizing vaccine that does not stop infection or transmission, that is driving breakthrough and variants…how to address this? you stop the vaccine program, the vaccine has failed as to effectiveness and it is harmful and you DO not vaccinate children with these non-sterilizing vaccines as they do not need it, have near zero risk, and can become super spreaders…we will damage their innate immune systems via the vaccinal antibodies and cause grave harm to warned…

Yes, it makes sense. Dont you think?

‘But when a person becomes infected months after vaccination, the antibodies that respond come from a new and improved batch made by long-lived cells that carry a memory of the pathogen. When the body encounters the pathogen again, these memory cells are called back to duty and have a chance to refine the antibodies, providing better protection against subsequent infections.’