Increased risk of autoimmune diseases post COVID gene injection; Vojdani & Kharrazian report on "Antigenic cross-reactivity between SARS-CoV-2 and human tissue with a possible link to an increase in

by Paul Alexander

autoimmune diseases"; accumulating evidence of cross reaction between spike protein antibody and tissue proteins

The principle is that if proteins on the COVID virus (e.g. viral surface etc.) mimic human tissue proteins, then the antibodies induced by vaccines would potentially attack the human tissue and thus lead to auto-reactivity, auto-immune disease. In that regard it would make complete sense to study the autoimmune-inducing capacity of the antigens on the COVID virus. This was not done by Pfizer or Moderna and not mandated by FDA. Thus could it be that the present devastating effects of the COVID gene therapy injection vaccine up to including death, may be a result of viral antigenic mimicry with human tissue?

As reported by Vojdani et al. (https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7246018/), we know that ‘vaccine-induced autoimmunity from autoimmune cross-reactivity is associated with narcolepsy, Guillain-Barré syndrome, multiple sclerosis, demyelinating neuropathies, systemic lupus erythematosus, and postural orthostatic tachycardia syndrome in susceptible subgroups as reported by Segal and Shoenfeld [2]’.

 

The findings reported in this study raise very serious questions about auto-immunity and the possible role of the COVID gene injections in promoting auto-immunity via viral antigenic mimicry (similarity) with human tissue (self-like, self-mimicking, and all the variations etc.).

SOURCE:

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7246018/

 

Fig. 1 above:

(A) Reaction of anti-SARS-CoV-2 spike protein monoclonal antibody with human tissue antigens. (B) Reaction of anti-SARS-CoV-2 nucleoprotein monoclonal antibody with human tissue antigens.

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