'MSH3 Homology and Potential Recombination Link to SARS-CoV-2 Furin Cleavage Site'' Ambati & Brufsky et al.; points to a likely deliberate manufacture of COVID virus with furin cleavage site insert
by Paul Alexander
A BLAST search revealed that a 19 nucleotide portion of the SARS.Cov2 genome encompassing the furin cleavage site is a 100% complementary match to a codon-optimized proprietary sequence...key!
Very important paragraph:
‘Among numerous point mutation differences between the SARS-CoV-2 and the bat RaTG13 coronavirus, only the 12-nucleotide furin cleavage site (FCS) exceeds 3 nucleotides. A BLAST search revealed that a 19 nucleotide portion of the SARS.Cov2 genome encompassing the furing cleavage site is a 100% complementary match to a codon-optimized proprietary sequence that is the reverse complement of the human mutS homolog (MSH3).’
Pay close attention to this paper and what it is tell you! It is basically showing you that it is impossible for this virus to have been zoonotic, leaping from animal to human. At some point yes, it was in the bats or racoon dogs or pangolin etc. But they took it into the lab and created a monster with poison pills. They know who they are and what they did. I know who they are. You know many of them too.
“Among numerous point mutation differences between the SARS-CoV-2 and the bat RaTG13 coronavirus, only the 12-nucleotide furin cleavage site (FCS) exceeds 3 nucleotides. A BLAST search revealed that a 19 nucleotide portion of the SARS.Cov2 genome encompassing the furing cleavage site is a 100% complementary match to a codon-optimized proprietary sequence that is the reverse complement of the human mutS homolog (MSH3). The reverse complement sequence present in SARS-CoV-2 may occur randomly but other possibilities must be considered. Recombination in an intermediate host is an unlikely explanation. Single stranded RNA viruses such as SARS-CoV-2 utilize negative strand RNA templates in infected cells, which might lead through copy choice recombination with a negative sense SARS-CoV-2 RNA to the integration of the MSH3 negative strand, including the FCS, into the viral genome. In any case, the presence of the 19-nucleotide long RNA sequence including the FCS with 100% identity to the reverse complement of the MSH3 mRNA is highly unusual and requires further investigations.”